What is a frozen shoulder?

A frozen shoulder is a shoulder joint with significant loss of its range of motion in all directions. The range of motion is limited not only when the patient attempts motion, but also when the doctor attempts to move the joint fully while the patient relaxes. A frozen shoulder is also referred to as adhesive capsulitis.

What causes a frozen shoulder?

Frozen shoulder is the result of inflammation, scarring, thickening, and shrinkage of the capsule that surrounds the normal shoulder joint. Any injury to the shoulder can lead to frozen shoulder, including tendinitis, bursitis, and rotator cuff injury. Frozen shoulders occur more frequently in patients with diabetes, chronic inflammatory arthritis of the shoulder, or after chest or breast surgery. Long-term immobility of the shoulder joint can put people at risk to develop a frozen shoulder.

How is a frozen shoulder diagnosed?

A frozen shoulder is suggested during examination when the shoulder range of motion is significantly limited, with either the patient or the examiner attempting the movement. Underlying diseases involving the shoulder can be diagnosed with the history, examination, blood testing, and x-ray examination of the shoulder.

If necessary, the diagnosis can be confirmed when an x-ray contrast dye is injected into the shoulder joint to demonstrate the characteristic shrunken shoulder capsule of a frozen shoulder. This x-ray test is called arthrography. The tissues of the shoulder can also be evaluated with an MRI scan.

What conditions can mimic a frozen shoulder?

Inflammation of the shoulder joint (arthritis) or the muscles around the shoulder can cause swelling, pain, or stiffness of the joint that can mimic the range of motion limitation of a frozen shoulder.

Injury to individual tendons around the shoulder (tendons of the rotator cuff) can limit shoulder-joint range of motion, but usually not in all directions. Often during the examination of a shoulder with tendon injury (tendinitis or tendon tear), the doctor is able to move the joint with the patient relaxed beyond the range that the patient can on their own.

How is a frozen shoulder treated?

The treatment of a frozen shoulder usually requires an aggressive combination of antiinflammatory medication, cortisone injection(s) into the shoulder, and physical therapy. Without aggressive treatment, a frozen shoulder can be permanent.

Diligent physical therapy is often key and can include ultrasound, electric stimulation, range-of-motion exercise maneuvers, ice packs, and eventually strengthening exercises. Physical therapy can take weeks to months for recovery, depending on the severity of the scarring of the tissues around the shoulder.

It is very important for people with a frozen shoulder to avoid reinjuring the shoulder tissues during the rehabilitation period. These individuals should avoid sudden, jerking motions of or heavy lifting with the affected shoulder.

Sometimes frozen shoulders are resistant to treatment. Patients with resistant frozen shoulders can be considered for release of the scar tissue by arthroscopic surgery or manipulation of the scarred shoulder under anesthesia. This manipulation is performed to physically break up the scar tissue of the joint capsule. It carries the risk of breaking the arm bone (humerus fracture). It is very important for patients that undergo manipulation to partake in an active exercise program for the shoulder after the procedure. It is only with continued exercise of the shoulder that mobility and function is optimized.

Frozen Shoulder At A Glance

• Frozen shoulder is the result of scarring, thickening, and shrinkage of the joint capsule.
• Any injury to the shoulder can lead to frozen shoulder.
• A frozen shoulder is usually diagnosed during an examination.
• A frozen shoulder usually requires aggressive treatment.

REFERENCES:

Koopman, William, et al., eds. Clinical Primer of Rheumatology. Philadelphia: Lippincott Williams & Wilkins, 2003.

Kelley's Textbook of Rheumatology, W B Saunders Co, edited by Shaun Ruddy, et al., 2000.

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